カレントテラピー 32-4 サンプル

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カレントテラピー 32-4 サンプル

14 Current Therapy 2014 Vol.32 No.4330参考文献1) Peters J, Jurgensen A, Kloppel G:Ontogeny, differentiationand growth of the endocrine pancreas. Virchows Arch 436:527-538, 20002) Gregg BE, Moore PC, Demozay D, et al:Formation of ahuman β-cell population within pancreatic islets is set earlyin life. J Clin Endocrinol Metab 97:3197-3206, 20123) Meier JJ, Butler AE, Saisho Y, et al:Beta-cell replication isthe primary mechanism subserving the postnatal expansionof beta-cell mass in humans. Diabetes 57:1584-1594, 20084) Kohler CU, Olewinski M, Tannapfel A, et al:Cell cycle controlof β-cell replication in the prenatal and postnatalhuman pancreas. Am J Physiol Endocrinol Metab 300:E221-E230, 20115) Maedler K, Schumann DM, Schulthess F, et al:Aging correlateswith decreased beta -cell proliferative capacity andenhanced sensitivity to apoptosis. A potential role for Fasand Pancreatic duodenal homeobox-1. Diabetes 55:2455-2462, 20066) Cnop M, Hughes SJ, Igoillo-Esteve M, et al:The long lifespanand low turnover of human islet beta cells estimated bymathematical modelling of lipofuscin accumulation. Diabetologia53:321-330, 20107) Perl S, Kushner JA, Buchholz BA, et al:Significant humanbeta-cell turnover is limited to the first three decades of lifeas determined by in vivo thymidine analog incorporation andradiocarbon dating. J Clin Endocrinol Metab 95:E234-E239,20108) Mizukami H, Takahashi K, Inaba W, et al:Age-associatedchanges of islet endocrine cells and the effects of body massindex in Japanese. J Diabetes Invest 4:2014 In press(doi:10.1111/jdi.12118)9) Hanley SC, Austin E, Assouline -Thomas B, et al:β-Cellmass dynamics and islet cell plasticity in human type 2 diabetes.Endocrinology 151:1462-1472, 201010) Butler AE, Janson J, Bonner-Weir S, et al:Beta-cell deficitand increased beta-cell apoptosis in humans with type 2 diabetes.Diabetes 52:102-110, 200311) Saisho Y, Butler AE, Manesso E, et al:Beta-cell mass andturnover in humans. Effects of obesity and aging. DiabetesCare 36:111-117, 201312) Kou K, Saisho Y, Satoh S, et al:Change in β-cell mass inJapanese nondiabetic obese individuals. J Clin EndocrinolMetab 98:3724-3730, 201313) Kim H, Toyofuku Y, Lynn FC, et al:Serotonin regulatespancreatic beta cell mass during pregnancy. Nat Med 16:804-808, 201014) Rieck S, Kaestner KH:Expansion of beta -cell mass inresponse to pregnancy. Trends Endocrinol Metab 21:151-158, 201015) Butler PC, Meier JJ, Butler AE, et al:The replication of betacells in normal physiology, in disease and for therapy. NatureClin Pract Endocrinol Metab 3:758-768, 200716) Koyama M, Wada R, Sakuraba H, et al:Accelerated loss ofislet beta cells in sucrose-fed Goto-Kakizaki rats, a geneticmodel of non -insulin -dependent diabetes mellitus. Am JPathol 153:537-545, 199817) Karamohamed S, Demissie S, Volcjak J, et al;NHLBI FraminghamHeart Study:Polymorphisms in the insulin-degradingenzyme gene are associated with type 2 diabetes in menfrom the NHLBI Framingham Heart Study. Diabetes 52:1562-1567, 2003